By American Academy of Ophthalmology, Robert H. Rosa Jr. MD
Part four offers fabrics in components: half I, Ophthalmic Pathology; and half II, Intraocular Tumors: scientific points. half I makes use of a hierarchy that strikes from basic to precise to aid derive a differential prognosis for a particular tissue. half II is a compilation of chosen medical elements of value to the final ophthalmologist. Following half II are the yank Joint Committee on melanoma 2010 staging varieties for ocular and adnexal tumors.
Upon of completion of part four, readers will be capable to:
Describe a dependent method of realizing significant ocular stipulations according to a hierarchical framework of topography, sickness strategy, basic analysis and differential diagnosis
Summarize the stairs in dealing with ocular specimens for pathologic learn, together with acquiring, dissecting, processing, and marking tissues
Identify these ophthalmic lesions that point out systemic illness and are most likely existence threatening
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Additional resources for 2014-2015 Basic and Clinical Science Course (BCSC): Section 4: Ophthalmic Pathology and Intraocular Tumors
3, At 1 week, migrating epithelium and endothelium partially seal the wound; fibroblasts begin to migrate and supply collagen . 4, Fibroblast activity and collagen and matrix deposition continue . The endothelium , sealing the inner wound, lays down new Descemet membrane. 5, Epithelial regeneration is complete. Fibroblasts fill the wound with type I collagen and repair slows. 6, The final wound contracts. The collagen fibers are not parallel with the surrounding lamellae. The number of fibroblasts decreases .
The stroma of the caruncle is the only part of the conjunctiva that (like skin) also contains sebaceous glands and hair follicles (Fig 5- lE). See BCSC Section 2, Fundamentals and Principles of Ophthalmology, and Section 8, External Disease and Cornea, for further discussion. Congenital Anomalies Choristomas A choristoma is a benign, congenital proliferation ofhistologically mature tissue elements not normally present at the site of occurrence. This heterotopic congenital lesion results from normal tissue migrating to or remaining in an abnormal location during embryo gen esis (hence the derivation from the Greek word for "separated mass").
D·,c acid-Schiff (PAS) peno ,la, Alcian blue 0 a Aliza ri n red Colloidal iron td IS V, Qllaterial Stained: Color Example Nucleus: blue Cytoplasm: red Glycogen and proteoglycans: magenta Acid mucopolysaccharide: blue Calcium: red Acid mucopolysaccharide: blue Amyloid: orange, red-green dichroism Acid-fast organisms: red Fungal elements: black General tissue stain (Fig 3-2E) Descemet membrane (Fig 6-17E) Cavernous optic atrophy (Fig 15-10B) Band keratopathy Macular dystrophy (Fig 6-21C) Lattice dystrophy (Fig 6-23C, D) Atypical mycobacterium Fusarium (Fig 6-7B) Amyloid: purple, violet Bacteria positive: blue Bacteria negative: red Lattice dystrophy Bacterial infection Collagen: blue Granular dystrophy (Fig 6-22C) Red deposits Fleischer ring Lattice dystrophy Temporal artery elastic layer (Fig 15-6B) Band keratopathy (Fig 6-12C) Stain Hema t o Xy Specimen Handling • 31 Congo red Ziehl-Neelsen Gornori methenamine silver (GMS) Crystal violet Gram stain (tissue Brown & Brenn [B&Bl or Brown & Hopps [B&H] stain) Masson trichrome Perls Prussian blue Thioflavin T (ThT) Verhoeff-van Gieson (elastic stain) von Kassa Muscle: red Iron: blue Amyloid: fluorescent yellow Elastic fibers: black Calcium phosphate salts: black ..